Collaborative knowledge creation in the higher education academic library

Collaboration has become a core competency of the 21st century workforce. Thus, the need of collaboration is reshaping the academic library in higher education to produce competent future workforce. To encourage collaboration in the academic library, knowledge commons that integrate technology to infrastructure and system furniture are introduced. The article examines college students’ collaborative activities for knowledge creation at a university academic library via a survey, using the theory of organizational knowledge creation. It analyzed student group activities, based on the four types of activities in knowledge creation. A total of 385 undergraduate students completed the survey. The survey results indicated that the most frequent group activity is individual-oriented activity, followed by socialization activity, creating contents as a group, and group learning activity. However, when analyzed by frequent activities by the same users, the majority of users were doing all four activities, followed by individual-oriented activity only, and individual-oriented and socialization activities. The results revealed different trends in the engagement of the four knowledge creation activities between knowledge workplace and the academic library. Several implications to encourage collaborative activities are suggested

Intracellular lumen extension requires ERM-1-dependent apical membrane expansion and AQP-8-mediated flux

SUMMARY Many unicellular tubes such as capillaries form lumens intracellularly, a process that is not well understood. Here we show that the cortical membrane organizer ERM-1 is required to expand the intracellular apical/lumenal membrane and its actin undercoat during single-cell C.elegans excretory canal morphogenesis. We characterize AQP-8, identified in an ERM-1 overexpression (ERM-1[++]) suppressor screen, as a canalicular aquaporin that interacts with ERM-1 in lumen extension in a mercury-sensitive manner, implicating water-channel activity. AQP-8 is transiently recruited to the lumen by ERM-1, co-localizing in peri-lumenal cuffs interspaced along expanding canals. An ERM-1[++]-mediated increase in the number of lumen-associated canaliculi is reversed by AQP-8 depletion. We propose that the ERM-1-AQP-8 interaction propels lumen extension by translumenal flux, suggesting a direct morphogenetic effect of water-channel-regulated fluid pressure